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Triptorelin

GnRH Agonist (long-acting)

Also known as: Decapeptyl · Trelstar · D-Trp6-GnRH

A synthetic GnRH agonist that produces initial LH/FSH surge followed by pituitary desensitization, driving medical castration. Used in prostate cancer, endometriosis, & precocious puberty.

Typical Dose

3.75 mg IM monthly (prostate cancer); 0.1 mg daily SubQ (precocious puberty)

Route

Intramuscular or subcutaneous injection

Cycle

Ongoing (cancer); fixed term (puberty); clinical supervision required

Half-life

~3 hours (short-acting); depot formulations release over 1–3 months

Storage

As provided by manufacturer. Reconstitute per product instructions.

Overview

Triptorelin is a synthetic decapeptide GnRH analog with a D-tryptophan substitution at position 6, extending its half-life & pituitary receptor binding. Initial administration causes a surge in LH & FSH ('flare'), followed by downregulation of GnRH receptors & sustained suppression of gonadotropins & sex steroids — a paradoxical effect exploited for medical castration.

It is FDA approved for prostate cancer, precocious puberty, & endometriosis. In research peptide contexts, it is sometimes used for HPTA restart protocols after anabolic steroid use, though evidence for this application is limited.

Quick Start Guide

1

Triptorelin is a clinical drug administered by physicians. Not suitable for self-administration outside established medical protocols.

Research Indications

Prostate cancer (medical castration)

Most Effective

FDA approved. Reduces testosterone to castrate levels within 2–4 weeks.

Endometriosis & uterine fibroids

Most Effective

FDA approved. Suppresses estrogen, reducing endometrial implant growth.

Precocious puberty

Most Effective

Continuous GnRH agonism suppresses premature gonadal activation in central precocious puberty.

Research Protocols

Prostate cancer (standard)

Ongoing

Dose

3.75 mg

Frequency

Monthly IM depot injection

Route

IM

Side Effects & Safety

Common

  • Hot flashes
  • Sexual dysfunction (intended effect in cancer)
  • Osteoporosis with long-term use
  • Initial testosterone flare (first dose)

Uncommon

  • Depression
  • Injection site reactions

When to Stop

  • Vertebral metastases with spinal cord compression (flare risk)
  • Signs of allergic reaction

How to Reconstitute

1

Reconstitute per manufacturer microsphere depot instructions for clinical use.

Dosing math: Clinical product only. Self-administration is not appropriate for most indications.

Quality Indicators

Good — use as normal

  • Manufacturer-stated appearance

Discard immediately

  • Any deviation from manufacturer specification

What to Expect

Days 1–7 (flare)

Testosterone surges before receptor downregulation. Risk window for clinical conditions sensitive to androgen flare.

Week 2–4

Testosterone suppression begins. Castrate levels achieved by day 21–28 in most patients.

Community Insights

Self-reported. Reflects user experience, not clinical outcomes.

Research References

Triptorelin in advanced prostate cancer: a systematic review

Prostate Cancer & Prostatic Diseases · 2010

Systematic review confirming efficacy of triptorelin depot for androgen deprivation therapy in prostate cancer.

Verify what you have

Information on this page applies to pharmaceutical-grade peptides. Purity & identity of research-grade products vary. Certipep provides independent ESI-TOF-MS & HPLC analysis with a signed analytical report.

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